Inflammation contributes to the atherogenic role of intermittent hypoxia in apolipoprotein-E knock out mice.
type: Dataset
description: International audience
landing page: 0021-9150
keyword: atherosclerosis
keyword: dyslipidemia
keyword: inflammation
keyword: infoeu-reposemanticsarticle
keyword: intermittent-hypoxia
keyword: journal-articles
keyword: mesh-animals
keyword: mesh-anoxia
keyword: mesh-aortic-diseases
keyword: mesh-apolipoproteins-e
keyword: mesh-atherosclerosis
keyword: mesh-blood-pressure
keyword: mesh-cells-cultured
keyword: mesh-cholesterol-dietary
keyword: mesh-diet-high-fat
keyword: mesh-disease-models-animal
keyword: mesh-dyslipidemias
keyword: mesh-inflammation
keyword: mesh-inflammation-mediators
keyword: mesh-lipids
keyword: mesh-male
keyword: mesh-mice
keyword: mesh-mice-inbred-c57bl
keyword: mesh-mice-knockout
keyword: mesh-plaque-atherosclerotic
keyword: mesh-risk-factors
keyword: mesh-time-factors
keyword: mice
keyword: sdvmhepcsclife-sciences-q-biohuman-health-and-pathologycardiology-and-cardiovascular-system
keyword: sleep-apnea
issued: 2026-05-14T14:42:52.683405
modified: 2026-05-14T14:42:52.683410
language: en
contact point: Organization
  fn: CCSD

test_moissonnage_selune
type: Agent

Inflammation contributes to the atherogenic role of intermittent hypoxia in apolipoprotein-E knock out mice.
type: Distribution
format: HTML
access URL: https://inserm.hal.science/inserm-00786362
issued: 2026-05-14T14:42:52.692478
modified: 2026-05-14T14:42:52.631342